Event Title
Heterozygous Mutant Huntingting Promotes Cadmium Neurotoxicity via Impaired Metal Transport, Caspase Mediated Apoptosis, Erk and PKCd Dependent Oxidative Signaling Mechanisms: Relevance to Pathogenesis of Huntington’s Disease
Location
Science Center, Bent Corridor
Start Date
11-2-2018 6:15 PM
End Date
11-2-2018 6:50 PM
Poster Number
3
Keywords:
Huntington's Disease, Cadmium, neurodegeneration, neurotoxicity
Recommended Citation
Thomas, Morgan and Ryan, Emmaleth, "Heterozygous Mutant Huntingting Promotes Cadmium Neurotoxicity via Impaired Metal Transport, Caspase Mediated Apoptosis, Erk and PKCd Dependent Oxidative Signaling Mechanisms: Relevance to Pathogenesis of Huntington’s Disease" (2018). Celebration of Undergraduate Research. 44.
https://digitalcommons.oberlin.edu/cour/2018/posters/44
Major
Morgan Thomas, Neuroscience
Emmaleth Ryan, Undeclared
Award
Emmaleth Ryan, Science and Technology Research Opportunities for a New Generation (STRONG)
Document Type
Poster
Heterozygous Mutant Huntingting Promotes Cadmium Neurotoxicity via Impaired Metal Transport, Caspase Mediated Apoptosis, Erk and PKCd Dependent Oxidative Signaling Mechanisms: Relevance to Pathogenesis of Huntington’s Disease
Science Center, Bent Corridor